The
obesity epidemic is one of the largest threats to public health around the globe.
Obesity can cause heart disease, stroke, cancer, and even more life-threatening conditions. One of the major concerns of scientists and government
officials worldwide is finding the best way to decrease obesity on an international scale. To some
the answer is quite simple, all it takes is a shift in people’s dietary habits toward eating
more fruits and vegetables and less processed foods and sugars.
However, for many these changes are not so simple. Tasty treats and snacks attract
people with promises of good taste but come with an unhealthy amount of sugar, fat, and calories. But why do we make unhealthy choices if we are aware
of the deadly consequences? Dr. Jen Beshel, a biologist studying this
phenomenon at Loyola University Chicago and policymakers in Chile both provide possible solutions to this widespread problem.
Dr. Jen
Beshel studies Drosophila and their physical responses to obesity-linked
behavior. She has been focusing her research on the proteins associated with the neuronal
pathways that could cause obesity. She discovered that the lack of protein upd2
led to fullness and a decrease in hunger in Drosophila which ultimately
resulted in lower weight. Another protein, upd1 works to prevent
overconsumption in flies, so she disabled this protein to see how the flies
would react. Sure enough, the flies ate large amounts of food and eventually
became obese. Dr. Beshel’s evidence shows that there may be some neural
signaling or connectivity associated with behaviors that could lead to obesity.
According
to the article, “In Sweeping War on Obesity, Chile Slays Tony the Tiger” from
the New York Times written by Andrew Jacobs, Chile has seen a rapid rise in
obesity. Andrews claims that Chile had previously been struggling with
widespread malnourishment up until the late 1980s but has since been faced with
an obesity epidemic. To combat this issue, the Chilean government implemented
“marketing restrictions” on unhealthy brands, particularly those that advertise
to children. According to Andrews, these restrictions include removing cartoon
characters from children’s cereal boxes, eliminating Kinder Surprise (a
chocolate egg that contains a toy inside), banning unhealthy foods from being
sold at Chilean schools, and getting rid of ads that promote unhealthy foods
for kids. The government is also testing out a new labeling system that places
black stop sign stickers on foods that are high in sugar, salt, calories, or
saturated fat. Due to these harsh restrictions, food companies have been
altering their products to contain less harmful ingredients, and schools have
started selling more nuts, rice cakes, and dried fruit. While it may take a few
years to see a decrease in obesity, the Chilean government is hopeful that
these new laws will improve the health of its citizens.
The
reasons behind obesity-linked behavior may still remain unclear. However, it is
critical that drastic steps be taken to improve the lives of those around the
world struggling with obesity. Dr. Beshel has found promising evidence that
supports the neurological pathways behind behaviors that lead to obesity. The Chilean government went
to extremes to provide a possible solution to the rising obesity rates of its
citizens. Through proper education, restriction, and research there will
hopefully be a decline in obesity around the globe and an increase in better
health for all.
Jacobs, Andrew. “In Sweeping War on Obesity, Chile Slays
Tony the Tiger.” The New York Times, The New York Times, 7 Feb. 2018, www.nytimes.com/2018/02/07/health/obesity-chile-sugar-regulations.html?rref=collection/sectioncollection/world&action=click&contentCollection=world®ion=stream&module=inline&version=latest&contentPlacement=9&pgtype=sectionfront.
Beshel, Jennifer, et al. A Leptin Analog Locally Produced in the Brain Acts via a Conserved Neural Circuit to Modulate Obesity-Linked Behaviors in Drosophila. 10 Jan. 2017, https://www.sciencedirect.com/science/article/pii/S1550413116306465?via%3Dihub.
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