Do Your Taste Buds Play a Role in Your Eating Habits?
Taste is one of six special senses other than: hearing, equilibrium, olfaction, vision and touch. In the article Ephrin-B/EphB Signaling Is Required for Normal Innervation of Lingual Gustatory Papillae, Dr. Rochlin, explains that although taste axons are ultimately restricted to taste bud related epithelium, they can also reside adjacent to non-taste epithelium. There is a possibility that non-diffusible guidance cues may have played a role in preventing nerve innervation of non-taste bud epithelium which is adjacent to these taste receptors. The research article looks at EphBs and how their expression in taste axons and that ephrin-B/EphB signaling is required for normal innervation of fungiform papillae of the tongue. Fungiform papillae are scattered on the tip of the tongue on to the sides and they have the nerve innervations to differentiate between the five tastes: sweet, sour, salty, sweet, and umami. Dr. Rochlin’s study has also found that Ephrin-B/EphB is sufficient enough to repel or suppress gustatory neuronal process from their cell body projection in vitroand are essential for gustatory papillae innervation shown in knockout mice model in vivoexperiment. Overall the study suggests that the quantity and quality of different competing taste signals have an influence on axon targeting outcomes, and provides good evidence that an ephrin is expressed in lingual gustatory epithelium, Eph-Bs are in taste axons, and ephrin-B/EphB signaling is needed for proper nerve innervation of fungiform papillae. In the article, How Sugar and Fat Trick the Brain into Wanting more Food, talks about how eating just for the sake of pleasure, although is not a new phenomenon, rather a very old unhealthy habit. However, new research has shown that there is more to pleasure eating and it has to do with how our taste buds respond to food that are particularly high in fats and sweets. These types of food somehow play a part in inducing some chemical change in brain which leads to overeating. This relatively new study has given a name to food craving as hedonic hunger, which is a powerful desire for food even if you’re not hungry. There is a very likely chance that in the U.S., hedonic hunger may have played a significant role in the increase of obesity given a very easy access to junk food. Although the classical view on hunger and weight regulation is based mainly on metabolism and maintaining homeostasis which are driven by physiological needs for energy required to function. Hypothalamus is a brain region known to regulate hunger and appetite and also production of hormones to regulate temperature, thirst, hunger, sleep, and mood. However, as it turns out that extremely sweet or fatty foods change the brain's reward circuit in the same way that cocaine can do. The brain begins responding to fatty and sugary food by simply looking at them before they even enter our mouth. As soon as such a dish touches the tongue, taste buds send signals to the hypothalamus of the brain, which in turn responds by spewing the neurochemical dopamine. And this cascade effect makes a person more addicted to such foods that are high in sweet and fats.
Taste is one of six special senses other than: hearing, equilibrium, olfaction, vision and touch. In the article Ephrin-B/EphB Signaling Is Required for Normal Innervation of Lingual Gustatory Papillae, Dr. Rochlin, explains that although taste axons are ultimately restricted to taste bud related epithelium, they can also reside adjacent to non-taste epithelium. There is a possibility that non-diffusible guidance cues may have played a role in preventing nerve innervation of non-taste bud epithelium which is adjacent to these taste receptors. The research article looks at EphBs and how their expression in taste axons and that ephrin-B/EphB signaling is required for normal innervation of fungiform papillae of the tongue. Fungiform papillae are scattered on the tip of the tongue on to the sides and they have the nerve innervations to differentiate between the five tastes: sweet, sour, salty, sweet, and umami. Dr. Rochlin’s study has also found that Ephrin-B/EphB is sufficient enough to repel or suppress gustatory neuronal process from their cell body projection in vitroand are essential for gustatory papillae innervation shown in knockout mice model in vivoexperiment. Overall the study suggests that the quantity and quality of different competing taste signals have an influence on axon targeting outcomes, and provides good evidence that an ephrin is expressed in lingual gustatory epithelium, Eph-Bs are in taste axons, and ephrin-B/EphB signaling is needed for proper nerve innervation of fungiform papillae. In the article, How Sugar and Fat Trick the Brain into Wanting more Food, talks about how eating just for the sake of pleasure, although is not a new phenomenon, rather a very old unhealthy habit. However, new research has shown that there is more to pleasure eating and it has to do with how our taste buds respond to food that are particularly high in fats and sweets. These types of food somehow play a part in inducing some chemical change in brain which leads to overeating. This relatively new study has given a name to food craving as hedonic hunger, which is a powerful desire for food even if you’re not hungry. There is a very likely chance that in the U.S., hedonic hunger may have played a significant role in the increase of obesity given a very easy access to junk food. Although the classical view on hunger and weight regulation is based mainly on metabolism and maintaining homeostasis which are driven by physiological needs for energy required to function. Hypothalamus is a brain region known to regulate hunger and appetite and also production of hormones to regulate temperature, thirst, hunger, sleep, and mood. However, as it turns out that extremely sweet or fatty foods change the brain's reward circuit in the same way that cocaine can do. The brain begins responding to fatty and sugary food by simply looking at them before they even enter our mouth. As soon as such a dish touches the tongue, taste buds send signals to the hypothalamus of the brain, which in turn responds by spewing the neurochemical dopamine. And this cascade effect makes a person more addicted to such foods that are high in sweet and fats.
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